Principles of Virology. Jane Flint. Читать онлайн. Newlib. NEWLIB.NET

Автор: Jane Flint
Издательство: John Wiley & Sons Limited
Серия:
Жанр произведения: Биология
Год издания: 0
isbn: 9781683673583
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from infected birds. The frightening possibility that it could gain the ability to spread among humans is a major incentive for monitoring for person-to-person transmission in case of infection by this and other pathogenic avian influenza viruses. Given the eons over which viruses have had the opportunity to interact with various species, today’s “natural” host may simply be a way station in viral evolution.

      Because viruses are dependent on their hosts for propagation, studies that focus on viral reprogramming of cellular mechanisms have provided unique insights into genetics, cellular biology, and functioning of host defenses. Groundbreaking studies of viruses that infect bacteria (called bacteriophages) in the mid-20th century established the molecular basis of genetic inheritance. Through development and use of stringent, quantitative methods with these relatively simple biological entities, this research confirmed that DNA encodes genes and genes encode proteins. General mechanisms of genetic recombination, repair, and control of gene expression were also elucidated, laying the foundations of modern molecular biology and recombinant DNA technology. Subsequent studies of animal viruses established many fundamental principles of cellular function, including the presence of intervening sequences in eukaryotic genes. The study of cancer (transforming) viruses established the genetic basis of this disease.

      With the development of recombinant DNA technology and our increased understanding of viral systems, it has become possible to use viral genomes as vehicles for the delivery of genes to cells and organisms for both scientific and therapeutic purposes. The use of viral vectors to introduce genes into various cells and organisms to study their function has become a standard method in biology. Viral vectors are also being used to treat human disease, for example, via “gene therapy,” in which functional genes delivered by viral vectors compensate for faulty genes in the host cells (Volume II, Chapter 9).

      Although viruses have been known as distinct biological entities for only about 120 years, evidence of viral infection can be found among the earliest recordings of human activity, and methods for combating viral disease were practiced long before the first virus was recognized. Consequently, efforts to understand and control these important agents of disease began only in the last century.

      Reconstruction of the prehistoric past to provide a plausible account of when or how viruses established themselves in human populations is challenging. However, extrapolating from current knowledge, we can deduce that some modern viruses were undoubtedly associated with the earliest precursors of mammals and coevolved with humans. Other viruses entered human populations only recently. The last 10,000 years of history was a time of radical change for humans and our viruses: animals were domesticated, the human population increased dramatically, large population centers appeared, and commerce and technology drove worldwide travel and interactions among unprecedented numbers of people.

      Viruses that established themselves in human populations were undoubtedly transmitted from animals, much as still happens today. Early human groups that domesticated and lived with their animals were almost certainly exposed to different viruses than were nomadic hunter/gatherer societies. Similarly, as many different viruses are endemic in the tropics, human societies in that environment must have been exposed to a greater variety of viruses than societies established in temperate climates. When nomadic groups met others with domesticated animals, human-to-human contact could have provided new avenues for virus spread. Even so, it seems unlikely that viruses such as those that cause measles or smallpox could have entered a permanent relationship with small groups of early humans. Such highly virulent viruses, as we now know them to be, either kill their hosts or induce lifelong immunity. Consequently, they can survive only when large, interacting host populations offer a sufficient number of naive and permissive hosts for their continued propagation. Such viruses could not have been established in human populations until large, settled communities appeared. Less virulent viruses that enter into a long-term relationship with their hosts were therefore more likely to be the first to become adapted to reproduction in the earliest human populations. These viruses include the modern retroviruses, herpesviruses, and papillomaviruses.