Vascular Medicine. Thomas Zeller. Читать онлайн. Newlib. NEWLIB.NET

Автор: Thomas Zeller
Издательство: Ingram
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Жанр произведения: Медицина
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isbn: 9783131768513
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      Brekenfeld C, Remonda L, Nedeltchev K, et al. Symptomatic intracranial haemorrhage after intra-arterial thrombolysis in acute ischaemic stroke: assessment of 294 patients treated with urokinase. J Neurol Neurosurg Psychiatry 2007; 78: 280–5.

      Brekenfeld C, Schroth G, Mattle HP, et al. Stent placement in acute cerebral artery occlusion: use of a self-expandable intracranial stent for acute stroke treatment. Stroke 2009; 40: 847–52.

      del Zoppo GJ, Poeck K, Pessin MS, et al. Recombinant tissue plasminogen activator in acute thrombotic and embolic stroke. Ann Neurol 1992; 32: 78–86.

      Furlan A, Higashida R, Wechsler L, et al. Intra-arterial proUK for acute ischemic stroke. The PROACT II study: a randomized controlled trial. Prolyse in Acute Cerebral Thromboembolism. JAMA 1999; 282: 2003–11.

      Gralla J, Brekenfeld C, Mordasini P, Schroth G. Mechanical thrombolysis and stenting in acute ischemic stroke. Stroke 2012; 43: 280–5.

      Hacke W, Kaste M, Bluhmki E, et al. Thrombolysis with alteplase 3 to 4.5 hours after acute ischemic stroke. N Engl J Med 2008; 359: 1317–29.

      IMS Study Investigators. Combined intravenous and intra-arterial recanalization for acute ischemic stroke: the Interventional Management of Stroke Study. Stroke 2004; 35: 904–11.

      Lewandowski CA, Frankel M, Tomsick TA, et al. Combined intravenous and intra-arterial r-TPA versus intra-arterial therapy of acute ischemic stroke: Emergency Management of Stroke (EMS) Bridging Trial. Stroke 1999; 30: 2598–605.

      Mattle HP, Arnold M, Georgiadis D, et al. Comparison of intra-arterial and intravenous thrombolysis for ischemic stroke with hyperdense middle cerebral artery sign. Stroke 2008; 39: 379–83.

      Mattle HP, Arnold M, Lindsberg PJ, Schonewille WJ, Schroth G. Basilar artery occlusion (Review). Lancet Neurol 2011; 11: 1002–14.

      Nedeltchev K, Arnold M, Brekenfeld C, et al. Pre- and in-hospital delays from stroke onset to intra-arterial thrombolysis. Stroke 2003; 34: 1230–4.

      Nedeltchev K, Brekenfeld C, Remonda L, et al. Internal carotid artery stent implantation in 25 patients with acute stroke: preliminary results. Radiology 2005; 237: 1029–37.

      Neumann-Haefelin T, du Mesnil de Rochemont R, Fiebach JB, et al. Effect of incomplete (spontaneous and postthrombolytic) recanalization after middle cerebral artery occlusion: a magnetic resonance imaging study. Stroke 2004; 35: 109–14.

      Ogawa A, Mori E, Minematsu K, et al. Randomized trial of intra-arterial infusion of urokinase within 6 hours of middle cerebral artery stroke: the middle cerebral artery embolism local fibrinolytic intervention trial (MELT) Japan. Stroke 2007; 38: 2633–9.

      Rha JH, Saver JL. The impact of recanalization on ischemic stroke outcome: a meta-analysis. Stroke 2007; 38: 967–73.

      Shaltoni HM, Albright KC, Gonzales NR, et al. Is intra-arterial thrombolysis safe after full-dose intravenous recombinant tissue plasminogen activator for acute ischemic stroke? Stroke 2007; 38: 80–4.

      

      The National Institute of Neurological Disorders and Stroke rt-PA Stroke Study Group. Tissue plasminogen activator for acute ischemic stroke. N Engl J Med 1995; 333: 1581–7.

      Theron J, Courtheoux P, Casasco A, et al. Local intra-arterial fibrinolysis in the carotid territory. AJNR Am J Neuroradiol 1989; 10: 753–65.

      Tomsick T, Broderick J, Carrozella J, et al. Revascularization results in the Interventional Management of Stroke II trial. AJNR Am J Neuroradiol 2008; 29: 582–7.

      Zeumer H, Hacke W, Ringelstein EB. Local intra-arterial thrombolysis in vertebrobasilar thromboembolic disease. AJNR Am J Neuroradiol 1983; 4: 401–4.

      Zeumer H, Hündgen R, Ferbert A, et al. Local intra-arterial fibrinolytic therapy in inaccessible internal carotid occlusion. Neuroradiology 1984; 26: 315–7.

      2 Thoracic arteries

      2.1 Arteriosclerotic and acquired inflammatory and congenital diseases of the thoracic aorta

      Anatomy of the whole aorta: Reinhard Putz

      Clinical findings: Friedhelm Beyersdorf and Thomas Zeller

      Conservative treatment: Friedhelm Beyersdorf and Thomas Zeller

      Endovascular treatment: Friedhelm Beyersdorf

      Surgical treatment: Friedhelm Beyersdorf

      Fig. 2.1–1 Overview of the whole aorta.

      The body’s main artery, the aorta, is divided into three parts (Fig. 2.1-1): the ascending aorta, the arch of the aorta and the descending aorta. The ascending aorta starts with the slightly dilated aortic bulb at the aortic valve, which with its three semilunar cusps (valvules) prevents backflow of blood during diastole (Fig. 2.1-2). The cusps consist of very firm, taut connective tissue and are covered with endothelium on both surfaces. They are attached to the inner wall of the junction between the left ventricle of the heart and the aorta and have small nodules (lunules) on their free edges. When blood is flowing back toward the heart, the cusps fill up (aortic sinus), pressing the lunules against each other and usually completely blocking backflow. These delicate edges interlock so finely that even slight changes due to various causes can unfortunately lead to insufficiency.

      In the area of the aortic bulb, two arteries supplying the heart are typically already given off from the ascending aorta. The right coronary artery arises from the wall of the right sinus of the aortic valve, and the left coronary artery from the left sinus (Fig. 2.1-2).

      Fig. 2.1–2 The aortic valve from above, with the origins of the coronary arteries.

      Fig. 2.1–3 Origins of the large arteries from the aortic arch (from Lippert and Pabst 1985).

      The ascending aorta does not give off any branches along its subsequent course. It passes into the arch of the aorta without a clear boundary. The upper margin of the aortic arch projects onto the manubrium of the sternum. It has an oblique angle and passes dorsally into the descending aorta, lying in the inferior posterior mediastinum. Normally (in 70% of cases), three large vessels arise from the arch of the aorta in a cranial direction, with considerable variation (Fig. 2.1-3). With the exception of branches to the chest, the entire right half of the head and right arm are supplied by the brachiocephalic trunk, which divides after a few centimeters into the right subclavian artery and right common carotid artery.

      The left common carotid artery and left subclavian artery are given off separately on the left.

      The most frequent variation is a separate origin, as early as at the aortic arch, of the two large arteries on the right side. Not infrequently, the right subclavian artery arises as the last branch from the aortic arch and then courses as the arteria lusoria behind the esophagus to the right side, where it branches further in the normal fashion. (This variant may seem surprising, but it is clearly explained by the development of the branchial arches.)

      The subclavian artery leaves the deep cervical region on each side, lying on the first rib behind the scalenus anterior. The common carotid artery courses upward in a common